MindMap Gallery chest wall pleural disease
This is a mind map about chest wall and pleural diseases, mainly including congenital chest wall deformities, empyema, chest wall tuberculosis, etc. Friends in need should quickly collect it!
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chest wall pleural disease
congenital chest wall deformity
pectus excavatum
Introduction
The sternum and ribs are sunken backward and inward, forming a funnel deformity
The deepest depression is at the junction of the sternum body and xiphoid process
clinical manifestations
Lateral chest X-ray → the lower sternum is depressed posteriorly
restrictive ventilatory dysfunction
treat
sternum lift
sternal flip surgery
Pedicle sternal flip surgery
chicken breast
Empyema
Introduction
Purulent infection in which purulent exudate accumulates in the pleural cavity
Pathogenic types are divided into
Purulent
Tuberculous
Specific pathogenicity
Pathological etiology
Mostly from infection focus in the lungs (common factor)
Empyema secondary to sepsis/septicemia
Mostly caused by blood group dissemination
Empyema caused by Staphylococcus aureus gradually increases
It is especially common in children
route of infection
Directly invades the pleural cavity from the suppurative lesion
via lymphatic route
Hematogenous dissemination
installment
Stage 1 (exudation stage) (parapneumonic effusion stage)
Profuse oozing of pus
Pus is thin and serous
Low white blood cells
No growth of pathogenic microorganisms
If drainage can be effective in the first stage → lung tissue can easily recruit
Stage II (purulent fibrin stage)
serous to purulent
Increased mesocytosis and fibrin
In the early stage, the cellulose is not firm and easy to fall off.
The abscess cavity tends to become localized (the pleural fluid protein divides the pleural fluid into multiple small cavities)
Stage three (chronic organizing stage)
Cellulose continues to thicken → scar tissue forms in the visceral wall
Limited lung expansion
acute empyema
clinical manifestations
Increased white blood cells
Chest tightness, cough and phlegm
The tremor is reduced on the affected side and dullness is detected by percussion.
The image shows a curved shadow that is high on the outside and low on the inside.
diagnosis
If a large amount of fluid accumulates
Large shadow on the diseased side Displacement of the diseased side of the mediastinum
Preferred examination → Ultrasound (to clarify the scope and location of empyema) (helpful for puncture treatment)
Confirmatory examination→Thoracentesis
chronic empyema
Cause
Improper treatment of acute empyema
Infection in the abscess cavity is difficult to control
pathology
Fibrous thickening of the parietal layer of the visceral pleura
Dense and thick wall → limited lung expansion
Respiratory function is affected
Difficulty breathing
Clubbing of fingers
The walls of the abscess cavity shrink → the mediastinum shifts to the diseased side
Pleural fibrosis/scar contraction
Parietal pleura thickening → Intercostal space narrowing → Thoracic collapse
Performance
restrictive respiratory dysfunction
treat
Pleural fiber stripping (main method)
Remove abscess cavity, parietal pleura and visceral pleura surface fiberboard → lung recruitment
Indications (early stage of chronic empyema)
Contraindications
The medical history is too long/the fiberboard is too thick
The lungs have shrunk for too long/the lung tissue has become severely fibrotic
Extensive inflammation/tuberculous cavities in the lungs
thoracoplasty
Purpose: Remove local hard tissue of the thoracic cage → invaginate the chest wall → eliminate the dead space between the two layers of pleura
pleural pneumonectomy
chest wall tuberculosis
pathology
dumbbell abscess
clinical manifestations
Systemic symptoms are not obvious
Cold abscesses (mostly abscesses that are not painful/hot/red)
If the abscess breaks through the skin → it discharges odorless, cloudy and cheese-like fluid
Replenish
Displacement to the healthy side
Acute empyema (fluid on the affected side)
Shift to the affected side
Chronic empyema (fibrotic traction on the affected side)