Source of infection

way for spreading

Population is susceptible

After HAV invades, it causes a brief viremia, then invades the liver and reproduces in hepatocytes. The virus invades the intestinal lumen from the biliary tract and is finally excreted in the feces.

Strengthen the management of food, water and feces

Vaccination

Hepadnaviridae

Source of infection

way for spreading

Population is susceptible

Mainly related to immune damage

active immunity

passive immunity

Source of infection

way for spreading

Population is susceptible

immune damage direct damage

Strict screening of blood donors

Strengthen blood product management

defective RNA virus

Source of infection

way for spreading

Population is susceptible

Infectious form

immune damage direct damage

Strict screening of blood donors

Strengthen blood product management

Source of infection

way for spreading

Population is susceptible

immune damage direct damage

Strengthen food, water and manure management

Vaccination

acute icteric hepatitis

acute anicteric hepatitis

If the disease lasts for more than half a year, it is divided into mild, moderate and severe.

Clinical manifestations: serum bilirubin>17 micromol/L, PTA<40%, hepatic encephalopathy

Classification

Severe jaundice that lasts for more than three weeks

Three separations

Obstructive

compensated cirrhosis

decompensated cirrhosis

health history

Physical examination

laboratory tests

Activity intolerance: related to impaired liver function and energy metabolism disorder

Malnutrition: lower than body requirements: related to loss of appetite, vomiting, diarrhea, digestive and absorptive dysfunction

Anxiety: related to repeated worries about the prognosis of the disease

Potential complications: hemorrhage, hepatic encephalopathy