MindMap Gallery Table of Contents of Cell Signal Transduction (Basics)
The 4th edition of "Cell Signaling Transduction (Basics)" welcomes learners in need, providing readers with a clear framework to help quickly understand the main chapters and topics of the book.
Edited at 2025-01-18 16:10:56Rumi: 10 dimensions of spiritual awakening. When you stop looking for yourself, you will find the entire universe because what you are looking for is also looking for you. Anything you do persevere every day can open a door to the depths of your spirit. In silence, I slipped into the secret realm, and I enjoyed everything to observe the magic around me, and didn't make any noise. Why do you like to crawl when you are born with wings? The soul has its own ears and can hear things that the mind cannot understand. Seek inward for the answer to everything, everything in the universe is in you. Lovers do not end up meeting somewhere, and there is no parting in this world. A wound is where light enters your heart.
Chronic heart failure is not just a problem of the speed of heart rate! It is caused by the decrease in myocardial contraction and diastolic function, which leads to insufficient cardiac output, which in turn causes congestion in the pulmonary circulation and congestion in the systemic circulation. From causes, inducement to compensation mechanisms, the pathophysiological processes of heart failure are complex and diverse. By controlling edema, reducing the heart's front and afterload, improving cardiac comfort function, and preventing and treating basic causes, we can effectively respond to this challenge. Only by understanding the mechanisms and clinical manifestations of heart failure and mastering prevention and treatment strategies can we better protect heart health.
Ischemia-reperfusion injury is a phenomenon that cellular function and metabolic disorders and structural damage will worsen after organs or tissues restore blood supply. Its main mechanisms include increased free radical generation, calcium overload, and the role of microvascular and leukocytes. The heart and brain are common damaged organs, manifested as changes in myocardial metabolism and ultrastructural changes, decreased cardiac function, etc. Prevention and control measures include removing free radicals, reducing calcium overload, improving metabolism and controlling reperfusion conditions, such as low sodium, low temperature, low pressure, etc. Understanding these mechanisms can help develop effective treatment options and alleviate ischemic injury.
Rumi: 10 dimensions of spiritual awakening. When you stop looking for yourself, you will find the entire universe because what you are looking for is also looking for you. Anything you do persevere every day can open a door to the depths of your spirit. In silence, I slipped into the secret realm, and I enjoyed everything to observe the magic around me, and didn't make any noise. Why do you like to crawl when you are born with wings? The soul has its own ears and can hear things that the mind cannot understand. Seek inward for the answer to everything, everything in the universe is in you. Lovers do not end up meeting somewhere, and there is no parting in this world. A wound is where light enters your heart.
Chronic heart failure is not just a problem of the speed of heart rate! It is caused by the decrease in myocardial contraction and diastolic function, which leads to insufficient cardiac output, which in turn causes congestion in the pulmonary circulation and congestion in the systemic circulation. From causes, inducement to compensation mechanisms, the pathophysiological processes of heart failure are complex and diverse. By controlling edema, reducing the heart's front and afterload, improving cardiac comfort function, and preventing and treating basic causes, we can effectively respond to this challenge. Only by understanding the mechanisms and clinical manifestations of heart failure and mastering prevention and treatment strategies can we better protect heart health.
Ischemia-reperfusion injury is a phenomenon that cellular function and metabolic disorders and structural damage will worsen after organs or tissues restore blood supply. Its main mechanisms include increased free radical generation, calcium overload, and the role of microvascular and leukocytes. The heart and brain are common damaged organs, manifested as changes in myocardial metabolism and ultrastructural changes, decreased cardiac function, etc. Prevention and control measures include removing free radicals, reducing calcium overload, improving metabolism and controlling reperfusion conditions, such as low sodium, low temperature, low pressure, etc. Understanding these mechanisms can help develop effective treatment options and alleviate ischemic injury.
Table of Contents of "Cell Signal Transduction (Basics)"
Book information
Edition: 4th edition
Editor-in-chief: Sun Daye, Cui Sujuan, Sun Ying
Publisher: Science Publisher
Chapter 1 Introduction
1. Research objects for cell signal transduction
2. Research scope and definition of cell signal transduction
3. The pathways and characteristics of cell signaling
4. Significance of cell signal transduction research
Main references
Chapter 2 Intercellular Signaling
1. Types of cell-to-cell communication
2. Types and characteristics of chemical signals secreted by cells
3. Hormones
4. Neurotransmitters, neuropeptides and chemokines
5. Cytokines and growth factors
6. Extracellular gases and small molecule signals
7. Intercellular signaling molecules in plants
Main references
Chapter 3 Receptor Types and Structures
1. Receptor overview
2. Types and structure of cell surface receptors
3. Nuclear receptors and cell surface receptors of steroid hormones
4. Internalization of cell surface receptors
5. Receptor research methods
Main references
Chapter 4 G protein-mediated signaling
1. Overview
2. Heterotrimer G protein
3. Small G protein superfamily
4. Plant G protein
5. Plant G protein
6. Commonly used research methods
Main references
Chapter 5: The Second Messenger System of Cyclonucleotides
1. Overview
2. Generation and inactivation of intracellular messenger cAMP
3. cAMP downstream target protein
4.cAMP signaling model
5. Cellular response regulated by cAMP signaling
6. Intracellular messenger cGMP
7. Plant cyclic nucleotide messenger system
8. Research method of cyclic nucleotide messenger system
Main references
Chapter 6 Phospholipid Signal System
1. Overview of plasma membrane phospholipids
2. Structural composition of plasma membrane phospholipids and metabolism of inositol phospholipids
3. Phospholipase C (PLC) and dual messenger signaling pathways
4. Phospholipase D (PLD)/phosphatidic acid (PA) signal
5. Phospholipase A (PLA)/lysophospholipid and free fatty acid signals
6. Plant phospholipase
7. Common research methods for lipid molecular metabolism
Main references
Chapter 7 Calcium Signal System
1. Overview of calcium signal
2. Generation of calcium signal and its regulatory factors
3. Termination of calcium signal
4. The space-time diversity of calcium signals
5. Calcium-binding protein
6. Calcium calmodulin-dependent protein phosphorylation
7. Cellular function regulated by calcium and calmodulin signal
8. Plant cell calcium signaling system
9. Research methods for calcium signaling
Main references
Chapter 8 Enzyme-active receptors
1. Overview of Enzyme Active Receptors
2. Tyrosine kinase receptor (RTK)
3.Serine/threonine kinase receptor (RSTK)
4. Guanylate cyclase-sodium urinary hormone peptide receptor (GC-NPR)
5. Protein tyrosine phosphatase receptor (RPTP)
6. Receptor-like kinases in plants
Main references
Chapter 9 The main recruitment receptors
1. Integrin receptors and focal adhesion kinase/cytoskeleton signaling pathways
2.Toll receptor/Toll-like receptor and NF-κB pathway
3. Cytokine receptors and JAK-STAT signaling pathways
4. Tumor necrosis factor receptor and its downstream signaling pathways
5. T cell receptors and their mediated signaling pathways
Main references
Chapter 10: Reversible protein phosphorylation modification and regulation of gene transcriptional activity
1. Overview
2. Phosphorylation of protein kinases and proteins
3. Dephosphorylation of protein phosphatase and protein
4. Regulation of gene transcriptional activity by reversible protein phosphorylation
5. Research methods for protein phosphorylation and dephosphorylation
Main references
Chapter 11 Other ways of modification and signal transduction of proteins
1. Overview
2. Methylation and demethylation
3.Acetylation and deacetylation
4. Ubiquitination and deubiquitination
5. Lipidization and delipidation
6. Glycosylation and deglycosylation
Main references
Chapter 12 Networking of Cell Signal Pathways
1. Interrelated relationships between cell signaling pathways
2. Formation of cellular signaling networks
3. Molecular basis for the formation of specificity of cytoplasmic signal networks
4. The molecular basis for the formation of the specificity of the signal pathway network in the nuclear
5. Research methods of cell signal transduction network
Main references
Appendix Noun abbreviation and Chinese-English comparison
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