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MindMap Gallery Mind Map: Mechanism of Action of CVS Drugs

Mind Map: Mechanism of Action of CVS Drugs

This mind map, created using EdrawMax, provides a detailed overview of the mechanisms of action of cardiovascular system (CVS) drugs. It categorizes drugs into cardiac glycosides, beta blockers, calcium channel blockers, diuretics, nitrates, and ACE inhibitors. Each category is further broken down into specific drugs and their mechanisms, such as how cardiac glycosides increase intracellular calcium concentration and how beta blockers reduce heart rate and contractility. The map serves as a comprehensive guide for understanding the pharmacological actions of CVS drugs.

Edited at 2025-07-10 15:59:58

Ainne Bhatti

Recent works View more works>>

Mind Map: Mechanism of Action of CVS Drugs
This mind map, created using EdrawMax, provides a detailed overview of the mechanisms of action of cardiovascular system (CVS) drugs. It categorizes drugs into cardiac glycosides, beta blockers, calcium channel blockers, diuretics, nitrates, and ACE inhibitors. Each category is further broken down into specific drugs and their mechanisms, such as how cardiac glycosides increase intracellular calcium concentration and how beta blockers reduce heart rate and contractility. The map serves as a comprehensive guide for understanding the pharmacological actions of CVS drugs.

Mind Map: Mechanism of Action of CVS Drugs

Ainne Bhatti

Recent works View more works>>

Mind Map: Mechanism of Action of CVS Drugs
This mind map, created using EdrawMax, provides a detailed overview of the mechanisms of action of cardiovascular system (CVS) drugs. It categorizes drugs into cardiac glycosides, beta blockers, calcium channel blockers, diuretics, nitrates, and ACE inhibitors. Each category is further broken down into specific drugs and their mechanisms, such as how cardiac glycosides increase intracellular calcium concentration and how beta blockers reduce heart rate and contractility. The map serves as a comprehensive guide for understanding the pharmacological actions of CVS drugs.

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Mechanism of Action of CVS Drugs

Cardiac glycosides

Digoxin

Increases intracellular calcium concentration

Enhances cardiac contractility

Inhibits Na+/K+ ATPase pump

Leads to increased intracellular sodium

Promotes calcium influx via Na+/Ca2+ exchanger

Induces vagal tone increase

Decreases heart rate

Reduces conduction velocity through AV node

Beta blockers

Nonselective beta blockers

Block both β1 and β2 adrenergic receptors

Decrease heart rate and contractility

Can cause bronchoconstriction

Used in hypertension and heart failure

Selective beta1 blockers

Primarily block β1 adrenergic receptors

Reduce heart rate and myocardial oxygen demand

Decrease renin release from kidneys

Used in hypertension and arrhythmias

Calcium channel blockers

Dihydropyridines

Primarily affect vascular smooth muscle

Cause vasodilation

Reduce systemic vascular resistance

Decrease blood pressure

Nondihydropyridines

Affect both myocardium and vascular smooth muscle

Reduce heart rate and contractility

Useful in managing angina and arrhythmias

ACE inhibitors

Inhibit angiotensinconverting enzyme

Decrease formation of angiotensin II

Promotes vasodilation

Reduces aldosterone secretion

Lower blood volume and blood pressure

Used in heart failure and hypertension

Provides renal protective effects

Improves outcomes postmyocardial infarction

Diuretics

Thiazide diuretics

Inhibit sodium reabsorption in distal convoluted tubule

Increase excretion of sodium and water

Decrease blood volume and blood pressure

Useful in hypertension and edema

Loop diuretics

Inhibit sodium and chloride reabsorption in loop of Henle

More potent than thiazides

Used in heart failure and acute pulmonary edema

Result in significant diuresis

Nitrates

Nitroglycerin

Converted to nitric oxide in the body

Causes relaxation of vascular smooth muscle

Leads to venodilation and reduced preload

Relieves angina symptoms

Improves blood flow to myocardium

Longacting nitrates

Used for chronic angina management

Administered at intervals to prevent tolerance

Enhance myocardial oxygen supply

Summary of mechanisms

Cardiac glycosides increase contractility and decrease heart rate

Beta blockers reduce heart rate and myocardial oxygen demand

Calcium channel blockers cause vasodilation and affect cardiac contractility

ACE inhibitors lower blood pressure and have renal protective effects

Diuretics reduce blood volume and lower blood pressure

Nitrates improve myocardial blood flow and relieve angina symptoms