MindMap Gallery Chapter 19 Heart Valvular Disease
The second chapter of Internal Medicine, Circulatory System Diseases, Valvular Heart Disease, VHD, summarizes knowledge on the etiology, pathophysiology, and clinical manifestations of mitral stenosis MS, mitral regurgitation MR, aortic stenosis AS, and aortic regurgitation AI. .
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This is a mind map about bacteria, and its main contents include: overview, morphology, types, structure, reproduction, distribution, application, and expansion. The summary is comprehensive and meticulous, suitable as review materials.
This is a mind map about plant asexual reproduction, and its main contents include: concept, spore reproduction, vegetative reproduction, tissue culture, and buds. The summary is comprehensive and meticulous, suitable as review materials.
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Valvular Heart DiseaseVHD
Mitral stenosisMS
Cause
rheumatic fever
It mostly occurs in young and middle-aged women aged 20-40 years old, and about half of the patients have no history of acute rheumatic fever.
degenerative disease
other
① Connective tissue disease ② Infectious endocarditis ③ Trauma ④ Congenital malformation
pathology
Diaphragm type
funnel type
Pathophysiology
Left atrial function compensation period
Left atrial decompensation
Right heart involvement and right heart failure stage
clinical manifestations
symptom
Dyspnea - exertional dyspnea, resting dyspnea, orthopnea, paroxysmal nocturnal dyspnea, acute pulmonary edema
Hemoptysis
Massive hemoptysis
severe distenosis
blood in sputum
paroxysmal nocturnal dyspnea
Coughing pink frothy phlegm
acute pulmonary edema
cough
Related to bronchial mucosal edema, pulmonary congestion, and atrial enlargement compressing the left main bronchus
Thromboembolism
other
Compression of the left recurrent laryngeal nerve causing hoarseness
Compression of the esophagus causing difficulty swallowing
physical signs
See
Severe bistenosis often has a mitral valve appearance
Precordial bulge
Right ventricular enlargement may show diffuse apical beats in the precordial area or enhanced cardiac fluctuations under the xiphoid process.
touch
Palpable diastolic tremor in apical region
knock
The heart expands to the left relative to the dullness boundary, giving a pear-shaped heart
listen
The S1 at the apex becomes hyperactive and beats like, and the valve opening sound can be heard. If the valve leaflets are calcified and stiff, the S1 weakens and the valve opening sound disappears.
In pulmonary hypertension, S2 in the pulmonary valve area is hyperactive or accompanied by division.
Mid to late diastolic rumble-like murmur was heard in the apical area
Pulmonary artery dilatation causes relative pulmonary valve insufficiency, and an early diastolic blow-like murmur is heard in the second intercostal space on the left sternal border.
The right ventricle was enlarged, and a holosystolic murmur was heard in the tricuspid valve area.
complication
Arrhythmia
Atrial fibrillation, induced acute pulmonary edema
acute pulmonary edema
Serious complications of severe bistenosis
Thromboembolism
right heart failure
Most common complications and causes of death from valvular heart disease
infective endocarditis
lung infection
Differential diagnosis
left atrial myxoma
tumor flutter
relativity dichotomy
Severe mitral regurgitation, congenital heart disease with massive left-to-right shunting, high circulatory dynamics
aortic valve insufficiency
Mid to late diastolic rumble-like murmur was heard in the apical area
Tuberculosis and bronchiectasis
treat in principle: Maintain and improve cardiac compensatory function, Limit physical activity, Prevent and treat streptococcal infections, Prevent recurrence of rheumatic fever, Prevent and treat complications
Management of complications
acute pulmonary edema
Atrial fibrillation: Medications to control ventricular rate
Prevention of embolism: long-term oral anticoagulant therapy with warfarin
Right heart failure: control sodium intake and use diuretics
Massive hemoptysis: Immediately take a seat, apply sedatives, and inject intravenous diuretics to reduce pulmonary venous pressure.
Mitral regurgitationMR
Cause
rheumatic fever
degenerative disease
Coronary heart disease, systemic lupus erythematosus, infective endocarditis
physical signs
See
Jugular vein distention, hepatojugular reflux sign ( ), and lower limb edema can be seen in right heart failure.
The apical beat is hyperdynamic and shifted downward to the left
touch
palpable lifting apical pulse
knock
The heart boundary expands to the lower left
listen
In patients with rheumatic heart disease, S1 in the apical area is weakened. In patients with mitral valve prolapse and coronary heart disease, S1 is often normal and S2 is divided and widened.
MR heart murmurs of different causes have different properties
rheumatic heart disease
A coarse holosystolic wind-like murmur of grade 3/6 or above is heard in the apex area, which is conducted to the left armpit and subscapular area. The inhalation is weakened, the expiration is slightly enhanced, and may be accompanied by tremor.
Mitral valve prolapse
Midsystolic click followed by late systolic murmur
Coronary heart disease papillary muscle dysfunction
Holosystolic murmur
Rupture of chordae tendineae
Noise like seagulls or musical sounds
In severe reflux, a short diastolic rumble-like murmur after S3 is heard in the apical area.
complication
atrial fibrillation
infective endocarditis
arterial thromboembolism
heart failure
Differential diagnosis
tricuspid valve insufficiency
ventricular septal defect
Aortic stenosis and pulmonary stenosis
Systolic ejection murmurs are heard in the second intercostal space on the right edge of the sternum and the second intercostal space on the left edge of the sternum.
hypertrophic obstructive cardiomyopathy
Aortic stenosis AS
Cause
degenerative disease
rheumatic fever
Congenital aortic valve single leaflet, double leaflet or triple leaflet malformation
Pathophysiology
The adult aortic valve orifice area is 3-4cm2.
When the valve orifice area decreases ≤1cm2, left ventricular systolic pressure increases significantly, transvalvular pressure difference increases, compensatory left ventricular hypertrophy, followed by left ventricular enlargement, reduced compliance, decompensation and left heart failure, and cardiac failure Reduced blood output
clinical manifestations
symptom
drowsy
Exertional dyspnea, paroxysmal nocturnal dyspnea, orthopnea, acute pulmonary edema
Angina pectoris
Fainting
physical signs
See
Increased and diffuse apical pulse
touch
In patients with obvious left ventricular hypertrophy, the apical pulse is shifted to the lower left and touches the lifting-like apical pulse.
In severe stenosis, carotid artery pulse is significantly delayed
Systolic tremor palpable in the second intercostal space at the right sternal border
knock
The border of cardiac dullness expands to the lower left
listen
S1 is normal, A2 is weakened, disappeared or reversely divided, and a (4-5)/grade 6 ejection systolic murmur can be heard in the aortic valve, which is rough, blowing, increasing-decreasing, and conducts to the neck or the left lower edge of the sternum.
Aortic regurgitation AI
physical signs
cardiac signs
See
The apical beat was hyperdynamic, expanded in scope, and shifted to the left and downwards
touch
The apex beats like a lift, expands in scope, and shifts to the left and downwards
knock
The border of cardiac dullness expands to the lower left, forming a boot-shaped heart
listen
S1 weakens and A2 weakens or disappears
An early diastolic sigh-like murmur was heard in the 2-3 intercostal spaces on the left sternal border and in the aortic valve area.
Late diastolic rumbling murmur heard at the apex
peripheral vascular sign
Increased pulse pressure difference
Nodding sign
Water rushing pulse, capillary pulsation sign ( )
gunshot sound, biphasic vascular murmur
complication
infective endocarditis
heart failure
Arrhythmia
The normal mitral valve orifice area is 4-6cm2 ① Mild stenosis 1.5-2 ② Moderate stenosis 1-1.5 ③Severe stenosis <1