MindMap Gallery Jaundice to be checked
This is a mind map about jaundice to be examined, and its main contents include: diseases of jaundice and fever, classification, and definition. The introduction is detailed and the description is comprehensive. I hope it will be helpful to interested friends!
Edited at 2025-01-04 14:05:07Rumi: 10 dimensions of spiritual awakening. When you stop looking for yourself, you will find the entire universe because what you are looking for is also looking for you. Anything you do persevere every day can open a door to the depths of your spirit. In silence, I slipped into the secret realm, and I enjoyed everything to observe the magic around me, and didn't make any noise. Why do you like to crawl when you are born with wings? The soul has its own ears and can hear things that the mind cannot understand. Seek inward for the answer to everything, everything in the universe is in you. Lovers do not end up meeting somewhere, and there is no parting in this world. A wound is where light enters your heart.
Chronic heart failure is not just a problem of the speed of heart rate! It is caused by the decrease in myocardial contraction and diastolic function, which leads to insufficient cardiac output, which in turn causes congestion in the pulmonary circulation and congestion in the systemic circulation. From causes, inducement to compensation mechanisms, the pathophysiological processes of heart failure are complex and diverse. By controlling edema, reducing the heart's front and afterload, improving cardiac comfort function, and preventing and treating basic causes, we can effectively respond to this challenge. Only by understanding the mechanisms and clinical manifestations of heart failure and mastering prevention and treatment strategies can we better protect heart health.
Ischemia-reperfusion injury is a phenomenon that cellular function and metabolic disorders and structural damage will worsen after organs or tissues restore blood supply. Its main mechanisms include increased free radical generation, calcium overload, and the role of microvascular and leukocytes. The heart and brain are common damaged organs, manifested as changes in myocardial metabolism and ultrastructural changes, decreased cardiac function, etc. Prevention and control measures include removing free radicals, reducing calcium overload, improving metabolism and controlling reperfusion conditions, such as low sodium, low temperature, low pressure, etc. Understanding these mechanisms can help develop effective treatment options and alleviate ischemic injury.
Rumi: 10 dimensions of spiritual awakening. When you stop looking for yourself, you will find the entire universe because what you are looking for is also looking for you. Anything you do persevere every day can open a door to the depths of your spirit. In silence, I slipped into the secret realm, and I enjoyed everything to observe the magic around me, and didn't make any noise. Why do you like to crawl when you are born with wings? The soul has its own ears and can hear things that the mind cannot understand. Seek inward for the answer to everything, everything in the universe is in you. Lovers do not end up meeting somewhere, and there is no parting in this world. A wound is where light enters your heart.
Chronic heart failure is not just a problem of the speed of heart rate! It is caused by the decrease in myocardial contraction and diastolic function, which leads to insufficient cardiac output, which in turn causes congestion in the pulmonary circulation and congestion in the systemic circulation. From causes, inducement to compensation mechanisms, the pathophysiological processes of heart failure are complex and diverse. By controlling edema, reducing the heart's front and afterload, improving cardiac comfort function, and preventing and treating basic causes, we can effectively respond to this challenge. Only by understanding the mechanisms and clinical manifestations of heart failure and mastering prevention and treatment strategies can we better protect heart health.
Ischemia-reperfusion injury is a phenomenon that cellular function and metabolic disorders and structural damage will worsen after organs or tissues restore blood supply. Its main mechanisms include increased free radical generation, calcium overload, and the role of microvascular and leukocytes. The heart and brain are common damaged organs, manifested as changes in myocardial metabolism and ultrastructural changes, decreased cardiac function, etc. Prevention and control measures include removing free radicals, reducing calcium overload, improving metabolism and controlling reperfusion conditions, such as low sodium, low temperature, low pressure, etc. Understanding these mechanisms can help develop effective treatment options and alleviate ischemic injury.
Jaundice to be checked
definition
Increased bilirubin concentration in serum causes yellowing of sclera, skin, mucous membrane, other tissues and body fluids. Normal value is 1.7-17.1umol/l, 17.1-34.2umol/L recessive jaundice, >34.2umol/L dominant jaundice
Classification
Category 1
True jaundice
Pseudojaundice
Eat carotene-containing fruits, vegetables, or drugs such as mipalin, especially in patients with liver insufficiency or hypothyroidism, the process of absorbed carotene converted into vitamin A in the liver is disordered, resulting in retention and carrots. Binemia, but not high bilirubin in the blood
Classification by bilirubin properties
Jaundice mainly due to indirect bilirubin elevation
Jaundice mainly due to direct bilirubin elevation
Classify by lesion site
Prehepatic jaundice
Hepatic jaundice
Posthepatic jaundice
Classified by cause
Hepatocellular jaundice
Total bilirubin and direct bilirubin increase, straight/total > 0.2-0.5, urobilirubin increases, normal or mild increase of urobilirubin, and increased aminotransferase and biliminal enzymes
Hemolytic jaundice
Total bilirubin and indirect bilirubin increase, urobilirubin negative, urobilirubin increase, and normal aminotransferase
Cholestatic jaundice
Total bilirubin and direct bilirubin increase, >0.5, urobilirubin, urobilirubin decrease, aminotransferase and biliminal enzyme increase, cholesterol increase
Congenital non-hemolytic jaundice
Diseases of jaundice and fever
Liver and gallbladder factors
Viral hepatitis
Acute cholangitis
Cancer jaundice
Liver abscess
Non-hepatobiliary factors
Lobular pneumonia, sepsis
Malignant histocellular disease
Leptospirosis
Acute hemolysis