1. Symptoms Eye pain, photophobia, tearing, and vision loss.

2. Physical signs Ciliary congestion, retrocorneal deposits, anterior chamber flare, changes in anterior chamber cells, iris and pupil, changes in the lens, changes in the vitreous body and fundus.

ciliary congestion

Differentiation between conjunctival congestion and ciliary congestion

Keratic precipitates (KP)

aqueous flare

aqueous cell

changes in iris

Vitreous body and fundus changes

1. Complicated cataract-retrocapsular opacification of the lens Cataract extraction and intraocular lens implantation were performed when inflammation was controlled.

2. Secondary glaucoma - a. Inflammatory cells, exudation and tissue fragments directly block the chamber angle; b. Inflammation of the anterior mucosa or trabecular meshwork around the iris, abnormal aqueous humor circulation; c. Pupil atresia or membrane atresia, blocking the atrium. Water enters the anterior chamber from the posterior chamber Intraocular pressure-lowering drugs, peripheral iridotomy or filtering surgery, etc.

3. Low intraocular pressure and eye atrophy - repeated inflammation causes ciliary body detachment or atrophy, and aqueous humor secretion decreases Anti-infective treatment and systemic or local supportive therapy are given.

1. Acute conjunctivitis: The identification point lies in whether there is an inflammatory reaction in the eye. 2. Acute angle-closure glaucoma: The identification points are whether the intraocular pressure is elevated and whether there is an inflammatory reaction in the eye. 3. Intraocular tumors: can cause pseudohyphema, which can be identified by ocular B-ultrasound, CT and other examinations. 4. Panuveitis: In addition to anterior inflammation, there is also inflammation in the posterior segment of the eye.

Symptoms: The onset is insidious. Physical signs: 1. The base of the vitreous body and the pars plana of the ciliary body: snowbank-like changes, mostly at the lower part, occasionally visible in 360°. 2. Vitreous body: snowball-like opacity.

3. Anterior segment: mild to moderate Children: Severe acute iridocyclitis.

4. Retina: (1) Vasculitis, perivasculitis Periphery>Houji Perivasculitis > Vasculitis vein>artery (2) Edema, exudation, and bleeding

1. Complicated cataracts 2. Macular edema and macular degeneration: anti-inflammatory, swelling and absorption-promoting treatment are performed. If there is choroidal neovascularization, anti-VEGF treatment or PDT treatment is possible. 3.Proliferative retinovitreopathy If traction retinal detachment occurs, vitreoretinal surgery should be used.

Choroiditis and Retinochoroiditis: Sympathetic ophthalmia, Vogt-Koyanagi Harada disease

Retinitis: Ocular toxoplasmosis, cytomegalovirus infection, Behcet's disease

Retinal vasculitis: Polyarteritis nodosa, Wegener's granulomatosis, Behcet's disease, Eales' disease

1. Symptoms Depends on the type of inflammation and the location of the damage. In the early stages, there are often no symptoms or only a flash of light in front of the eyes. In severe cases, symptoms such as severe vision loss, foggy vision, solid scotoma in the central field of vision, visual field defects, and visual distortion occur.

2. Physical signs

clinical manifestations Imaging examinations: FFA, OCT, B-ultrasound, CT, MRI, etc.

Laboratory tests: Routine blood tests and biochemical tests, bacterial smears, virus isolation and bacterial culture, etc. Immune antigen and antibody detection: Serological testing, lymphocyte transformation test and leukocyte migration inhibition test.

Dilate pupils, antagonize inflammation, and eliminate the cause of disease 1. Cycloplegic agent: For acute and severe inflammation, use 1% to 2% atropine eye ointment. After 2 to 3 days, change to 2% homatropine eye ointment. When the inflammation is mild, tropicamide can be used. Eye drops can prevent posterior adhesion, relieve ciliary muscle and sphincter spasm, relieve pain, and promote the dissipation of inflammation.

2. Glucocorticoids: The type, dosage and frequency of eye drops should be determined according to the severity of inflammation. 3. Non-steroidal anti-inflammatory drugs: inhibit arachidonic acid products. Commonly used drugs include pranoprofen and diclofenac sodium. The frequency of drugs and eye drops should be determined according to the severity of inflammation. 4. Antibiotics: Sensitive antibiotics or antiviral drugs should be used systemically or locally if caused by infectious factors.

5. Immunosuppressants: People with recurrent inflammation, especially those with systemic lesions or when inflammation is difficult to control, may consider glucocorticoids combined with immunosuppressants. Commonly used immunosuppressants include chlorambucil, cyclosporine, When using cyclophosphamide, etc., attention should be paid to systemic side effects. 6. Other therapies: hot compress, scleral condensation, laser photocoagulation, vitrectomy.

Sudden onset, symptoms of meningeal irritation Severe central nervous system involvement A small number of mild mental disorders Temporary increase in cerebrospinal fluid pressure lymphocytosis

Symptoms in both eyes Symptoms: sharp decrease in vision, eye pain, photophobia, flash of light Distortion Papilledema and hyperemia, retinal edema, venous congestion, radial macular wrinkles, exudative retinal detachment Diffuse choroiditis, discitis, retinal neuroepithelial detachment

Anterior segment: acute exudative iridocyclitis Non-granulomatous anterior uveitis manifestations such as dusty KP, anterior chamber flare, and anterior chamber cells

Inflammation gradually subsides Most patients have posterior iris synechiae The fundus looks like sunset, with nodules D-F Skin depigmentation changes

Oral: Indomethacin Ibuprofen Eye drops: anti-inflammatory eye drops Diclofenac Sodium Eye Drops Auxiliary medication vitamins traditional Chinese medicine Local mydriasis is extremely important